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Equally critical is the identification of biomarkers capable of defining ASD subgroups in whom intervention with oxytocin, a synthetic analog or even an antagonist of OXTR, might yield beneficial outcomes, given the heterogeneity in ASD etiology, phenotype, and treatment response [112, 113]
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N-acetylcysteine supplementation decreases osteoclast differentiation and increases bone mass in mice fed a high-fat diet
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