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The presence of diabetes and hyperglycemia tends to shift monocytes towards a functionally proinflammatory phenotype, which induces chronic inflammation and insufficient M2 functional profibrotic TGF-β signaling [29] similar to that seen in G6PD-deficient subjects
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![PDF] The promoting effect of pentadecapeptide BPC 157 on tendon healing involves tendon outgrowth, cell survival, and cell migration](https://figures.semanticscholar.org/be2e9ad5a4e60d33e6684941aa34c686bb34873f/2-Figure1-1.png)
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