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Constitutively active form of GPCR increases the chance of phosphorylation by G protein-coupled receptor kinases (GRKs) or other kinases (cAMP-dependent PKA and lipid signaling-activated PKC), and facilitates the binding of β -arrestin to the phosphorylated receptor, blocking G protein interaction with a receptor [22–24]
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(2003) Replacement therapy for vitamin B12 deficiency: comparison between the sublingual and oral route
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