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Data from the in vivo mice trial revealed that although the secretion level of IFNγ was only higher in wild-type group compared to uninfected group, proinflammatory cytokines in the Δ BacA group were more comparable to wild-type group as both wild-type and Δ BacA groups had higher secretion of IP-10, MIG, and TNFα compared to uninfected groups

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