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when the nuclear glutathione level is high, the expression of this proliferative marker was also high, while the depletion of nuclear glutathione with DEM caused the important decrease in the level of this protein, and consequently, the decrease in the proliferation

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Site-directed mutagenesis experiments show that cysteine-553 in human GLCLC is involved in heterodimer formation between the catalytically active GLCLC and the auxiliary, activity-enhancing GLCLR unit, which likely occurs through a disulfide bond [139]