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Histone acetylation by histone acetyltransferases (HATs) is likely to activate transcription by relaxing chromatin and histone deacetylation by histone deacetylases (HDACs) is likely to inhibit transcription by compacting chromatin (90)

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However, because of uncertainty over whether changes in GR-dependent glutathione status are themselves sensed or rather affect gene expression through secondary effects on ROS availability, further work is required to resolve this issue (Mhamdi et al., 2010a)