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Interestingly, l-selenocysteine Se -conjugates are more active aminotransferase and β-lyase substrates of rat kidney GTK than are the corresponding cysteine S -conjugates, in most cases by an order of magnitude or more (Commandeur et al., 2000)

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Our data thus establish a link between GSH deficiency in diabetes and increased susceptibility to melioidosis that may open up new therapeutic avenues to protect diabetic patients against some intracellular bacterial pathogens